The risk of hypercortisolism is even greater in people with difficult-to-control T2D and hypertension1
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When people with difficult-to-control type 2 diabetes (T2D) aren’t reaching their blood glucose goal despite standard-of-care therapies, it’s time to consider a different underlying cause. Recent study results revealed that 24% had endogenous hypercortisolism.1,2
Treatment of comorbidities without addressing the underlying hypercortisolism may not reduce cardiovascular risk2,3
The 1-mg dexamethasone suppression test (DST) detects all etiologies of hypercortisolism2
References
1. Buse JB, Kahn SE, Aroda VR, et al. Prevalence of hypercortisolism in patients with difficult-to-control type 2 diabetes: updated results from CATALYST part 1 [symposium]. Presented by Fonseca, V. at the 22nd World Congress Insulin Resistance Diabetes & Cardiovascular Disease; December 12-14, 2024; Los Angeles, CA. 2. DeFronzo RA, Auchus RJ, Bancos I, et al. BMJ Open. 2024;14(7):e081121. doi:10.1136/bmjopen-2023-081121 3. Petramala L, Olmati F, Concistrè A, et al. Endocrine. 2020;70(1):150-163. doi:10.1007/s12020-020-02297-2